甲亢的发病机制-自身免疫性疾病

    The disease must be of autoimmune disease, but its mechanism has not been fully elucidated. One of its features is the presence of serum and thyroid tissue can react or stimulate the role of autoantibodies, the antibody can stimulate the rodent animal thyroid, improve its function and cause tissue hyperplasia, but its role of slow and long-lasting.

关于治疗甲状腺病的医院
    Thus the name of thyroid stimulating immunoglobulin ( TSI ) or thyroid stimulating antibody ( TSAb ), clinically referred to as TSH receptor antibody ( TRAb ), as the disease lymphocyte secretion of IgG, Q and their corresponding antigens for TSH receptor or adjacent thyroid plasma membrane surface portion, when TSI and thyroid cell binding, TSH the receptor is activated, so that the thyroid function was stimulated, causing thyrotoxicosis and thyroid goiter, its role and effect of TSH resembles.

   国内甲亢治疗最佳医院
Now think of autoantibodies produced mainly with genetic defects associated with the inhibition of T lymphocytes ( Ts ) function to reduce the. The Ts function leads to defects in T helper cell inappropriate sensitized, and in May due to virus infection induced by interleukin 1 and interleukin 2 in the role of B cells to produce antisperm autoantibodies thyroid antibody. Separate immune monitoring defect is not sufficient to explain some of the specific immune lesions, also need to contact to the gene order coupling mechanism.